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These alterations create a vicious cycle of mitochondrial dysfunction, oxidative injury, and myofiber apoptosis, ultimately leading to irreversible supraspinatus atrophy and impaired regeneration after rotator cuff tears

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The goal is often to increase pathological response rates, potentially improving long-term survival

When this pathway breaks down it causes increased levels of oxidative stress within the cell that leads to glutathione depletion and mitochondrial dysfunction (49)

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doi: 10.3390/nu10101553 156 VelmuruganBSubapriyaRNaginiS